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Cell Systems Corporation human umbilical vein endothelial (huve) cells (mixed genders, pooled sources)
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Lonza huvecs lonza c2519as
β 3 AR-stimulated angiogenesis is NOS-dependent. (A) Tubule formation in HUVECs and the effect of l -NAME (300 μmol/L); n = 5 (B) cell migration rate in HUVECs and the effect of l -NAME (300 μmol/L); n = 4. (C) Tubule formation in human adult dermal microvascular <t>endothelial</t> cells (MVEC) and (D) patient-derived endothelial colony forming cells (ECFC) with β 3 AR agonist CL 316,243 (100 ng/ml) and β 3 AR antagonist, SR 592630 A (1 μmol/L) n = 3. (E) Representative images in ECFCs and (F) participant characteristics and medical history of ECFC source. Mean ± SEM; ** p < 0.01, **** p < 0.0001 vs control; # p < 0.05, ## p < 0.01, #### p < 0.0001 vs. CL 316,243 by 1-way ANOVA with Bonferroni post-hoc analysis or 2-way ANOVA. ACE/ARB, angiotensin converting enzyme inhibitor/angiotensin receptor blocker; BMI, body mass index; CACS, coronary artery calcium score; SPS, soft plaque score.
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Cambrex huvec cc-2517a
β 3 AR-stimulated angiogenesis is NOS-dependent. (A) Tubule formation in HUVECs and the effect of l -NAME (300 μmol/L); n = 5 (B) cell migration rate in HUVECs and the effect of l -NAME (300 μmol/L); n = 4. (C) Tubule formation in human adult dermal microvascular <t>endothelial</t> cells (MVEC) and (D) patient-derived endothelial colony forming cells (ECFC) with β 3 AR agonist CL 316,243 (100 ng/ml) and β 3 AR antagonist, SR 592630 A (1 μmol/L) n = 3. (E) Representative images in ECFCs and (F) participant characteristics and medical history of ECFC source. Mean ± SEM; ** p < 0.01, **** p < 0.0001 vs control; # p < 0.05, ## p < 0.01, #### p < 0.0001 vs. CL 316,243 by 1-way ANOVA with Bonferroni post-hoc analysis or 2-way ANOVA. ACE/ARB, angiotensin converting enzyme inhibitor/angiotensin receptor blocker; BMI, body mass index; CACS, coronary artery calcium score; SPS, soft plaque score.
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ATCC reference identifiers additional information cell line
β 3 AR-stimulated angiogenesis is NOS-dependent. (A) Tubule formation in HUVECs and the effect of l -NAME (300 μmol/L); n = 5 (B) cell migration rate in HUVECs and the effect of l -NAME (300 μmol/L); n = 4. (C) Tubule formation in human adult dermal microvascular <t>endothelial</t> cells (MVEC) and (D) patient-derived endothelial colony forming cells (ECFC) with β 3 AR agonist CL 316,243 (100 ng/ml) and β 3 AR antagonist, SR 592630 A (1 μmol/L) n = 3. (E) Representative images in ECFCs and (F) participant characteristics and medical history of ECFC source. Mean ± SEM; ** p < 0.01, **** p < 0.0001 vs control; # p < 0.05, ## p < 0.01, #### p < 0.0001 vs. CL 316,243 by 1-way ANOVA with Bonferroni post-hoc analysis or 2-way ANOVA. ACE/ARB, angiotensin converting enzyme inhibitor/angiotensin receptor blocker; BMI, body mass index; CACS, coronary artery calcium score; SPS, soft plaque score.
Reference Identifiers Additional Information Cell Line, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
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Image Search Results


β 3 AR-stimulated angiogenesis is NOS-dependent. (A) Tubule formation in HUVECs and the effect of l -NAME (300 μmol/L); n = 5 (B) cell migration rate in HUVECs and the effect of l -NAME (300 μmol/L); n = 4. (C) Tubule formation in human adult dermal microvascular endothelial cells (MVEC) and (D) patient-derived endothelial colony forming cells (ECFC) with β 3 AR agonist CL 316,243 (100 ng/ml) and β 3 AR antagonist, SR 592630 A (1 μmol/L) n = 3. (E) Representative images in ECFCs and (F) participant characteristics and medical history of ECFC source. Mean ± SEM; ** p < 0.01, **** p < 0.0001 vs control; # p < 0.05, ## p < 0.01, #### p < 0.0001 vs. CL 316,243 by 1-way ANOVA with Bonferroni post-hoc analysis or 2-way ANOVA. ACE/ARB, angiotensin converting enzyme inhibitor/angiotensin receptor blocker; BMI, body mass index; CACS, coronary artery calcium score; SPS, soft plaque score.

Journal: Frontiers in Pharmacology

Article Title: β 3 Adrenergic Receptor Stimulation Promotes Reperfusion in Ischemic Limbs in a Murine Diabetic Model

doi: 10.3389/fphar.2021.666334

Figure Lengend Snippet: β 3 AR-stimulated angiogenesis is NOS-dependent. (A) Tubule formation in HUVECs and the effect of l -NAME (300 μmol/L); n = 5 (B) cell migration rate in HUVECs and the effect of l -NAME (300 μmol/L); n = 4. (C) Tubule formation in human adult dermal microvascular endothelial cells (MVEC) and (D) patient-derived endothelial colony forming cells (ECFC) with β 3 AR agonist CL 316,243 (100 ng/ml) and β 3 AR antagonist, SR 592630 A (1 μmol/L) n = 3. (E) Representative images in ECFCs and (F) participant characteristics and medical history of ECFC source. Mean ± SEM; ** p < 0.01, **** p < 0.0001 vs control; # p < 0.05, ## p < 0.01, #### p < 0.0001 vs. CL 316,243 by 1-way ANOVA with Bonferroni post-hoc analysis or 2-way ANOVA. ACE/ARB, angiotensin converting enzyme inhibitor/angiotensin receptor blocker; BMI, body mass index; CACS, coronary artery calcium score; SPS, soft plaque score.

Article Snippet: Human umbilical vein endothelial cells (HUVECs; Lonza C2519AS, pooled source, Australia) were grown using standard cell culture conditions in endothelial cell growth medium (EGM plus ® , containing 2% fetal bovine serum, Lonza, Australia).

Techniques: Migration, Derivative Assay, Control